HMI discovers bacterial DNA can trigger Tau protein misfolding in Alzheimer’s disease models, potentially bringing new targets for therapeutic intervention

Researchers from Human Microbiology Institute (HMI) and Mitchell Center for Alzheimer's disease at the University of Texas McGovern Medical School in Houston have discovered that bacterial extracellular DNA can trigger Tau protein misfolding into neurotoxic aggregates, a key process in Alzheimer's disease pathogenesis. The discovery sets bacterial DNA as a new potential therapeutic target for treatment of ["]

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